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Neonatology , , 4 , Models of hypoxia and ischemiainduced seizures. Methods , , , A review of the conundrum of mild hypoxic-ischemic encephalopathy: Current challenges and moving forward.
Early Hum. Protein SB in umbilical cord blood as a potential biomarker of hypoxic-ischemic encephalopathy in asphyxiated newborns. Targeted temperature management in neurological intensive care unit. World J Methodol. Management and investigation of neonatal encephalopathy: update. Child Fetal Neonatal Ed. Pediatr Neonatol. SB - a potential biomarker for early detection of neonatal brain damage following asphyxia.
Fetal Neonatal. Neonatal hypoxic ischemic encephalopathy: an update on disease pathogenesis and treatment. Expert Rev. Neonatal Netw. Erythropoietin: A novel therapy for hypoxic-ischaemic encephalopathy? Child Neurol.
Mymensingh Med. PLoS One , , 12 10 , e Human recombinant erythropoietin in asphexia neonatorum: Pilot trial. Pediatrics , , 5 , ee Magnesium therapy in birth asphyxia. Indian J. Part Pediatric advanced life support: American heart association guidelines for cardiopulmonary resuscitation and emergency cardiovascular care. Circulation , , 18 Suppl 3 , SS Washington, DC.
Transplant Proc. The relationship between umbilical cord arterial pH and serious adverse neonatal outcome: Analysis of 51, consecutive validated samples. BJOG , , , Mild hypoxic ischaemic encephalopathy and long term neurodevelopmental outcome - A systematic review.
Targeting sentinel proteins and extrasynaptic glutamate receptors: A therapeutic strategy for preventing the effects elicited by perinatal asphyxia? Vitamin D insufficiency in neonatal hypoxic-ischemic encephalopathy.
Status of vitamin D, antioxidant enzymes, and antioxidant substance in neonates with neonatal hypoxic- ischemic encephalopathy. Fetal Neonatal Med. Acute endotoxemia in mice induces down regulation of megalin and cubilin in the kidney. Kidney Int. Vitamin D homeostasis is compromised due to increased urinary excretion of the hydroxycholecalciferol-vitamin D-binding protein complex in the Zucker diabetic fatty rat.
Vitamin D deficiency exacerbates experimental stroke injury and dysregulates ischemiainduced inflammation in adult rats. Endocrinology, , , Vitamin D and DBP: the free hormone hypothesis revisited. Steroid Biochem. Elevation of serum S protein concentration as a marker of ischemic brain damage in extremely preterm infants. Serial protein S serum levels in preterm babies with perinatal asphyxia and periventricular white matter lesions. Umbilical cord blood concentrations of ubiquitin carboxy-terminal hydrolase L1 UCH-L1 and glial fibrillary acidic protein GFAP in neonates developing hypoxic-ischemic encephalopathy.
M; Sannia, A. PLoS One , , 10 1 , e Plasma biomarkers of brain injury in neonatal hypoxic-ischemic encephalopathy. An update on diagnostic and prognostic biomarkers for traumatic brain injury. Anticancer Res. Neonatal brain injury. Proinflammatory cytokines, enolase and S as early biochemical indicators of hypoxic-ischemic encephalopathy following perinatal asphyxia in newborns. Converging pathways lead to overproduction of IL in the absence of vitamin D signaling.
Mechanisms underlying effects of 1, Dihydroxyvitamin D3 on the Th17 cells. Bp , , 3 4 , Altered circulating leukocytes and their chemokines in a clinical trial of therapeutic hypothermia for neonatal hypoxic ischemic encephalopathy. Care Med. Hagag, Mohamed S. El Frargy and Amal E. Texto principal 2.
Neonatal hypoxic-ischemic encephalopathy HIE is the result of a global hypoxic-ischemic brain injury in a term neonate, usually after asphyxia. It is important to remember that neonatal encephalopathy may result from a variety of conditions and hypoxic-ischemic brain injury is the most important of them 1. Consequently, both terms are frequently used as synonyms. Hypoxic-ischemic encephalopathy is one of the most common causes of cerebral palsy and other severe neurological deficits in children, occurring in of every live births.
Sobrino Maldonado, Jesus
Molteno C, Lachman P. The a etiology oflearning disability in preschool children with special reference to preventability. Ann Trop Paediatr. The incidence and severity of post-asphyxia encephalopathy in full-term infants. Early Hum Dev. World Health Organization. Neonatal and perinatal mortality: country, regional and global estimates.
Vitamina D en la encefalopatía hipóxico isquémica neonatal
Neonatal hypoxic ischemic brain injuries can manifest in different patterns of involvement depending on the severity and timing of the insult. When considering the perinatal maturation process of the brain and the severity of an insult, it is possible to understand the various manifestations. Typical patterns relative to timing of insult are as follows, with each condition described in detail in separate articles. Please Note: You can also scroll through stacks with your mouse wheel or the keyboard arrow keys. Updating… Please wait. Unable to process the form. Check for errors and try again.